🤖 AI Summary
This paper addresses a fundamental question in causal mechanism identification: under what conditions can heterogeneous treatment effects (HTEs) be used to infer the activation of a specific causal mechanism? It highlights that prevailing HTE detection methods—relying on pre-treatment covariates—implicitly assume linearity or additivity, rendering them invalid for mechanism inference under nonlinear outcome generation.
Method: We formally characterize the necessary and sufficient conditions under which HTEs carry identifying information about mechanism activation. Using potential outcomes frameworks, mechanism identification theory, and HTE modeling, we prove that nonlinear transformations of outcomes generally eliminate the inferential value of HTEs for mechanisms.
Contribution: We derive testable experimental design principles and an interpretive framework that explicitly delineate the boundaries of mechanism identification. Our results provide empiricists with robust identification guidelines and a systematic pathway for sensitivity analysis, bridging theoretical causality and applied HTE estimation.
📝 Abstract
The credibility revolution advances the use of research designs that permit identification and estimation of causal effects. However, understanding which mechanisms produce measured causal effects remains a challenge. A dominant current approach to the quantitative evaluation of mechanisms relies on the detection of heterogeneous treatment effects with respect to pre-treatment covariates. This paper develops a framework to understand when the existence of such heterogeneous treatment effects can support inferences about the activation of a mechanism. We show first that this design cannot provide evidence of mechanism activation without an additional, generally implicit, assumption. Further, even when this assumption is satisfied, if a measured outcome is produced by a non-linear transformation of a directly-affected outcome of theoretical interest, heterogeneous treatment effects are not informative of mechanism activation. We provide novel guidance for interpretation and research design in light of these findings.